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IFN-I signaling promotes Chac2-mediated GSH reprogramming and N1-type polarization of neutrophils To identify upstream regulatory pathways responsible for the observed GSH metabolic reprogramming in BM neutrophils during periodontitis, we performed gene set enrichment analysis (GSEA) of neutrophil transcriptomes from Lig and Con mice

Retama monosperma

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2014;5:e1202

Notably, many tumors also express functional lipoprotein lipase and can harvest fatty acids from triglyceride-rich lipoproteins via two routes: (i) lipoprotein lipase -mediated extracellular hydrolysis with subsequent uptake (e.g., via the CD36 receptor) and (ii) a non-catalytic bridging mode in which the lipase tethered to heparan-sulfate proteoglycans interacts with very low density lipoprotein receptor family pathways to internalize lipoprotein particles 14,15,16,17,18,19