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[PubMed] [Google Scholar] Edelmann S., Fahrner R., Malinka T., Song B

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A study on human postmortem brain tissue has revealed that the pathological progression of AD is accompanied by decreased expression of nuclear receptor coactivator 4 and GPX4 in gray matter (86)

In distal tubular injury, leakage of urinary regulatory proteins into the interstitial space activates dendritic cells through TLR4 and NLRP3 inflammasomes, initiating an immune response [323, 324]

Furthermore, GPx3 deficiency in patients has been linked to increased OS, endothelial dysfunction, coronary thrombosis, and diminished left ventricular function [113, 114]