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Mechanistically, NNMT suppression increased adipose SAM and NAD, upregulated polyamine flux enzymes (ODC, SSAT), and altered histone methylation consistent with a shift toward oxidative metabolism [2]

Missense mutation in selenocysteine synthase causes cardio-respiratory failure and perinatal death in mice which can be compensated by selenium-independent GPX4

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Stochastic simulation of cellular metabolism

Decreased expression of HNF4 by miR-34a in turn decreases cAMP-responsive element binding protein H (CREBH), which impairs VLDL assembly and secretion, leading to lipid accumulation in hepatocytes and possibly contributing to hepatic steatosis [299]

H., Stanbury, D