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Furthermore, IgD - CD27 + switched memory B cell counts seem to be more affected in CPs than IgD + CD27 + unswitched memory B cell counts ( Figures 6D, E )

This has been validated with animal models, for example, PPAR knockout (KO) mice showed reduced FA and increased glucose oxidation, and were resistant to DCM development.28 Conversely, in hearts of diabetic mice that overexpress PPAR, higher myocardial triglyceride levels alongside a more severe DCM phenotype were observed.29 Fig

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