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doi:10.1111/ede.12457

Hormonal modulation of oxidative stress The hormonal imbalance centered on E not only promotes reactive oxygen species (ROS) production but also weakens the cell's antioxidant defense capabilities

This study validated CSE as a necessary factor for VEGF-mediated neovascularization in vivo

ROS are by-products of aerobic metabolism and their production is enhanced during stress conditions through the disruption of electron transport system, and oxidizing metabolic activities occurring in chloroplasts, mitochondria and microbodies [20]

Lithium and the glutamatergic pathway Lithium administration has been also associated with an influence on the levels of proapoptotic proteins

HNA-1: human non-mercaptoalbumin type 1