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These enzymes are widely distributed in nature and are involved in a wide variety of functions 1

Some studies show accelerated healing and improved structural organization of injured tissues compared with untreated controls.[5] Researchers propose several mechanisms: angiogenesis, collagen remodeling, and fibroblast recruitment

Indigestible fragments of gluten are able to bind CXCR3 and release zonulin.[13] Studies have shown that elevated serum zonulin levels increased in obese versus non-obese subjects, and there is evidence of a correlation between total bacteria and serum zonulin levels, suggesting that the gut microbiota may cause increased zonulin levels, and subsequent abnormal gut permeability to endotoxin and ultimately micro-inflammation has been reported in obesity.[14] A qualitative and quantitative imbalance in the composition of the intestinal microbiota or dysbiosis contributes to intestinal barrier dysfunction and leaky gut syndrome