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In this context, the silencing of AGPAT4, which normally converts LPA into phosphatidic acid, leads to elevated LPA levels that stimulate M1 macrophage polarization via LPAR1/3p38 MAPKNF-B signaling, increasing the release of proinflammatory cytokines (IL-1, IL-6, and TNF-) and facilitating CD4/CD8 T-cell infiltration and IFN- production, ultimately suppressing tumor growth

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Interim findings showed that anti-phosphorylated tau (pTau) IgG responses were robust across all dosage levels, with titres rapidly increasing and remaining stable after the boost