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Its activity on HDL likely contributes to HDLs anti-oxidative capacity, as inhibition of HDL-associated Lp-PLA 2 attenuated HDLs ability to block LDL oxidation (848)

However, these effects cannot adequately explain axon degeneration induced by cisplatin and oxaliplatin, which target cellular DNA

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PBP1b and LpoB form a transient complex Given that LpoB-mediated activation is critical for PBP1b catalysis in vivo, we sought to explore how LpoB contributes to each step of the polymerization reaction by measuring LpoB binding to PBP1b in the presence of lipid II substrate and glycan chain intermediates

Some patients experience ASCVD events despite achieving LDL-C goals, while others with apparently modest LDL-C elevations have high particle burden or inherited risk factors such as elevated Lp(a)