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PMID: 2911387

CDK2 destabilizes tumor suppressor C/EBP expression through ubiquitin-mediated proteasome degradation in acute myeloid leukemia

This structural divergence underpins its specialized functional properties: while it demonstrates partial overlap through synergistic interactions with other members (e.g., cooperating with C/EBP to drive adipogenesis via PPAR and collaborating with C/EBP in granulopoiesis [5]), C/EBP executes non-redundant roles in terminal differentiation of granulocytes and adipocytes and exhibits exclusive oncogenic functions [6]

[DOI] [PubMed] [Google Scholar] 111.Epis R, Marcello E, Gardoni F, Longhi A, Calvani M, Iannuccelli M, et al