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however, little is known about how oxidative stress may play a critical role in this condition (Hulsebosch et al., Evidence supports that after a central nervous system injury, increased extracellular glutamate levels activate several intracellular pathways including ROS formation

Modulation of N-methyl-D-aspartate receptor function by glycine transport

In another oxidative stress-induced apoptosis model, 6-Hydroxydopamine (6-OHDA) was used as an inducer in catecholaminergic PC-12 cells, and significant neuroprotective effects against apoptosis were reported, even better than the green tea polyphenol mixture [92]

Hydrogen peroxide-induced potentiation of contractile responses in isolated rat airways

Lephart ED, Naftolin F

Disturbed balance between phase I and II metabolizing enzymes in ovarian endometriosis: a source of excessive hydroxy-estrogens and ROS