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These results thus show that spike protein-dependent loss of CFTR, and rescue of lost CFTR by cardiac glycoside drugs, can be detected in epithelial cells other than lung, and in cells for which CFTR is recombinant rather than native

International immunopharmacology, 2013, 15(2): 240245

In experimental studies, AOD-9604 has been observed to stimulate fat oxidation , increase energy expenditure , and reduce adipose tissue accumulation through activation of beta-adrenergic lipolytic pathways

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Stay with them until the emergency services arrive

N., Tothy, P