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BPC 157-activated endothelial nitric oxide synthase (eNOS) is associated with tissue repair and angiogenesis as reported in previous studies
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Disturbances in components in the p53/MDM2/IGF-1R network may cause IGF-1R upregulation and growth advantage for the cancer cell
This concept encompasses various changes including decreased motility, increased DNA fragmentation, morphological abnormalities, and reduced fertilization capacity
