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Additionally, exendin-4 treatment mitigated high glucose-induced mitochondrial dysfunction, as evidenced by reduced expression levels of mitochondrial function-associated genes mitochondrial calcium uniporter (MCU) and uncoupling protein 3 (UCP3), as well as the mitochondrial fission-related gene dynamin-related protein 1 (DRP1) [79]

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Patel and Dr

doi:10.2174/1871530322666221004151212 78
The natural release of dopamine is altered to rely on the drug

While clinical trials have provided valuable insights, further research is should determine the optimal use of these agents in different heart failure populations
