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doi: 10.1113/jphysiol.2006.107391
Here, we show in both ex vivo and in vivo models of ischemic injury that treatment with GLP-1(2836), a neutral endopeptidasegenerated (NEP-generated) metabolite of GLP-1, was as cardioprotective as GLP-1 and was abolished by scrambling its amino acid sequence
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Based on the above discussed, patients with liver disease and DM may be benefited with incretin-based therapies due to their low liver toxicity and wide tolerance
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