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GLP-1 receptor agonists work by reducing appetite, increasing satiety, and slowing gastric emptying, which affects how quickly food leaves your stomach

Nielsen M, Andreatta M

The same thing is true with these medications

One possible explanation is that the anti-inflammatory and neuroimmune effects of GLP-1 receptor activation may occur at lower levels of receptor engagement than the metabolic effects (like weight loss or appetite suppression)

Studies involving female cohorts were considered when relevant for mechanistic insights but were not used to extrapolate clinical conclusions regarding TRT

It also appears unlikely that GLP1 receptormediated effects on cell survival via ERK1/2 signalling can be clearly separated from activation of cAMP/PKA pathways, not least as ERK1/2 phosphorylation is partly PKA dependent