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Given that P5, a G-protein-biased agonist with a severely blunted -arrestin response has less propensity to induce GLP-1R desensitization, sustained activation of the receptor in adipose tissue may lead to the changes we observed in eWAT

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Besides, 16S amplicon rRNA sequencing for bacterial identification has detection limitations at species or strain levels and some key microbial features remained unclassified

There is a need for further studies involving combination AOM regimens that include GLP-1 receptor agonists for obesity management

Additional subgroup- and sensitivity analyses yielded results similar to the main analysis

Reshoring API production would require a massive investment in public-private partnerships, government incentives, and regulatory reform