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Reactive oxygen species are involved in smoking-induced dysfunction of nitric oxide biosynthesis and upregulation of endothelial nitric oxide synthase: an in vitro demonstration in human coronary artery endothelial cells

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Vascular endothelial dysfunction is the first step in atherosclerosis and plays an important role in the development, maintenance, and progression of atherosclerosis, resulting in cardiovascular events [5, 6]