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Loss of neurons from laminas I-III of the spinal dorsal horn is not required for development of tactile allodynia in the spared nerve injury model of neuropathic pain

*Correspondence: Rahmatullah Jan, [email protected]
S.ToddR

[10] The modifications, however, were not regarded as clinically important

Akash AI, et al

This protection is primarily attributed to the activation of the expression level of PGC-1, which plays a crucial role in maintaining normal mitochondrial function [187]
