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5 Besides numerous clinical trials in progress and over dozens of molecules being engineered against VEGF/VEGF receptor (VEGFR) and non-VEGF pathways, 4, 6 the entire situation warrants the understanding of molecular underpinnings and loopholes supporting the bypass angiogenic mechanisms ensuring tumor progression and metastasis even after the treatment with effective anti-angiogenic agents
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MIC+B12 Methionine The essential amino acid works as a deactivating agent
