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Brain temperature and its role in physiology and pathophysiology: Lessons from 20 years of thermorecording

This genetic NF1 mouse models has shown that a lipid storage disease phenotype may underlie muscle weakness in NF1, thus displaying commonalities with the lipid storage myopathies (LSMs), which also present with progressive muscle weakness and muscle lipid accumulation, and may occasionally be treated with high dose L-carnitine supplementation [19]

Nat Med (2017) 23:10713

A More Complete Perspective Peptides can signal repair, but they dont replace the raw materials required for that repair to occur

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