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These may arise indirectly from rapid weight loss-induced metabolic shifts, including nutrient and electrolyte depletion (e.g., magnesium, potassium, B-vitamins, iron), mitochondrial energy stress, and release of stored fat-soluble toxins (environmental chemicals, pesticides) into circulation during adipose tissue breakdown

doi: 10.1038/s41598-022-05197-9

W., da Silva, L., Maciel, G., Huguenin, A

HDL-induced NO underlies many of HDLs beneficial properties to endothelial cells, including HDL-induced vasodilation, tightening of cell-to-cell junctions and increased barrier function, differentiation of endothelial progenitor cells, cell survival and proliferation, cell migration, inhibition of apoptosis, and suppression of adhesion molecule expression