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As discussed above, it is appealing to infer a direct connection to the capacity of ERK to promote the transition to a plasmablast/plasma cell fate via BACH2 [158, 160, 161, 211]

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Additional research has investigated neuroprotective mechanisms, including neuronal survival, peripheral nerve regeneration, and functional recovery following experimental neurological injury

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doi: 10.1292/jvms.18-0045