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As a result, a new ferroptosis inhibition axis with GTP cyclohydrolase-1 (GCH1) as the core was finally determined, which was found to be mainly regulated by the GCH1/BH4/DHFR pathway, which also protected cells from ferroptosis in a GPX4-independent manner ( Figure 1D )

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Because the mutation rate in cellular genes is thousands of times slower than that for an RNA virus, if there is a physical interaction between these two proteins as Gordon et al