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Typically administered via subcutaneous injection in experimental settings, it is now available in a breakthrough capsule formula with high oral bioavailability

Studies indicate that although ZO-1-deficient cells can maintain the structure of TJs and exhibit normal permeability, the activity of other TJ proteins such as occludin and claudins in assembling TJs was delayed in these cells [8, 15, 16]

Therefore, it was concluded that the peptide might act preferentially on prokaryotic but not on eukaryotic membranes

It is not a magic bullet and it will not do away with surgery or fix structural issues such as bone-on-bone arthritis, or reverse severe deformities