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Council, N
As a result, glutathione is at the forefront of mitigating quinone-derived toxicity, by enzymatically reducing redox-generated H 2 O 2 to harmless H 2 O via glutathione peroxidase, or by direct reaction and detoxification of the quinone electrophile (Fig
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Study on oxidative stress and antioxidant level in patients of acute myocardial infarction before and after regular treatment
New Microbes New Infect 42:100897
