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Mechanistically, TMAO may contribute to kidney disease pathogenesis by inhibiting autophagy, activating the NLRP3 inflammasome, and inducing mitochondrial dysfunction
Emiatt L-karnitin szint gyakran alacsonyabb vegetrinusok s vegnok krben, mivel k nem fogyasztanak llati eredet termkeket
hypokalemia is a negative predictor for survival
Other findings demonstrate that eNOS activity remains unchanged in the aorta of rats treated with TMAO and in HAEC pre-incubated with 1 M of TMAO [57]
