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Remarkably, PP2 altered the GSB-106-elicited Tyr 706/707 and Tyr 515 phosphorylation more profoundly than that induced by BDNF, whereas the decrease of Tyr 816 phosphorylation was less striking in case of GSB-106

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PMOS-related insulin resistance causes hyperinsulinaemia chronically elevated insulin that overstimulates androgen production in the ovaries

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