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DNMT1 can inhibit ferroptosis by activating the RUNX/p53 pathway and promoting the expression of SLC7A11
Indeed, several studies have been conducted on the subject
The role of cellular protection in response to ROS accumulation has been reported, and the signal of preconditioning that leads to this protection was related to Hormesis, which means a dose-response relationship of ROS, at low levels, generates a stimulus of neurodegenerative diseases, while its accumulation (high concentration) generates an inhibition of the effect through preconditioning [21-23]

The American Journal of Medicine (2013) 126, 141-149

Furthermore, Mishra et al

However, this dysregulation can be reversed by H 2 S donors (162)
