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mTORC2 remains unaffected and responds by upregulating Akt, driving signals through the inhibited mTORC1

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Therefore, it is necessary to further explore the precise metformin doses required to change the AMP/ATP ratio and activate AMPK, in order to fully understand the effect of metformin on complex I (Table 4)

This amino acid can serve as a respiratory substrate that enters the TCA cycle in mitochondria, thereby driving ATP production (Obrist et al., 2018)

in the treatment of hypertension and hyperlipidemia: a comprehensive review of animal and human studies