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Similarly, MSH presented positive results in a model of renal fibrosis, induced by the administration of cyclosporine A, including reduction in TGF-1, inflammatory markers, cell apoptosis and collagen deposition (192)
These include the CXCL family, IL8, CCL20, ANGPT4 or CD49, as well as the response to heat stress, TGF- and IL-3/IL-4 signaling [156]
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