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Long COVID: post-acute sequelae of COVID-19 with a cardiovascular focus
doi: 10.1093/nar/gks827 79 YangRGaoWWangZJianHPengLYuXet al
Skeletal muscle mitochondria dysfunction in genetic neuromuscular disorders with cardiac phenotype
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Key findings include: Upregulation of VEGFR2 expression and internalization in endothelial cells Activation of the VEGFR2-Akt-eNOS signaling cascade Enhanced endothelial cell proliferation and tube formation Improved collateral circulation in ischemic injury models The peptides pro-angiogenic effects appear particularly relevant in healing scenarios where blood supply restoration is critical for tissue recovery
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