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These substitutes significantly enhanced neural cell survival post-TBI.[102] Furthermore, the study indicated a reduction in inflammation and apoptosis in the brain tissue, suggesting a protective role of the IGF-1-releasing dural substitutes.[102] Building on this, another study showed that animals treated with exosomes postinjury exhibited faster neurological recovery.[104] Specifically, the exosome-treated animals demonstrated improved motor function, reduced brain lesion sizes, and enhanced synaptic plasticity, indicating the potential of exosomes in promoting neural repair.[104] A pediatric preclinical study on the effects of LM22A-4, a TrkB agonist, revealed that treated animals displayed improved anxiety-related behavior.[21] In addition, there was a notable reduction in myelin deficits, suggesting that LM22A-4 could aid in the repair of damaged neural pathways in pediatric TBI cases.[21] However, the PROTECT III and SYNAPSE studies, two important phase-III clinical trials, found that early administration of progesterone did not yield any benefits in terms of neurological recovery for TBI patients

The causes of ASD remain complex and unclear, and there are currently no clinical biomarkers for autism spectrum disorder

Abstract EpsteinBarr virus (EBV) is a carcinogenic -herpesvirus that remains latent in more than 95% of adults

Next, high-resolution TEM (HR-TEM) images indicated the amorphous nature of five nanozymes (Fig

Busetto L, Bettini S, Makaronidis J, et al