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Baseline inflammatory markers (CRP, ESR, IL-6 if available), imaging of known endometriotic lesions, a bone density scan if you have had prior GnRH agonist use, and metabolic parameters provide objective data to track whether the medication is producing measurable changes

[DOI] [PMC free article] [PubMed] [Google Scholar] 86.Fackelmann G., Sommer S

Traditional approaches like nonsteroidal anti-inflammatory drugs (NSAIDs) suppress inflammation broadly, which may actually impair long-term tissue healing

In other words, Keap1 is sensitive to oxidative stress and acts as an NRF2 molecular switch

Turck et al., 2016)

For example, mutations in the p53 tumor suppressor gene, often caused by oxidative stress, are frequently observed in various cancers [99]