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Gclc deficiency in mouse CNS causes mitochondrial damage and neurodegeneration

Activation of CMA hinders the endocytosis and degradation of epidermal growth factor receptor (EGFR), thereby activating the EGFR signaling pathway

Besse-Patin et al report that PGC1A promotes ER expression while also acting as an ER coactivator to mediate an antioxidant response involving increased expression of antioxidant genes such as superoxide dismutase 2 and glutathione peroxidase-1 (84)

Table 2 summarizes the crucial anticancer mechanisms associated with copper deficiency discussed above