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In addition to its role in the inflammatory phase of wound healing, Vitamin A will also strengthen the collagen bonds and reduce the ability of wound inhibitory enzymes to counteract the healing process

Hearts from heterozygous (GPx1 +/ ) knockout mice were more susceptible to diastolic dysfunction following ischemia/reperfusion (I/R) injury than wildtype hearts [39] and GPx1 / hearts (from male mice) showed both contractile and diastolic dysfunction following I/R, with an increase in oxidative stress as measured by an increase in protein carbonylation [40]

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MAB, a generally applicable molecular force field for structure modelling in medicinal chemistry

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