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This suggests the existence of an intracellular reduction system capable of regenerating selenomethionine from its sulfoxide form
Taha and colleagues specifically observed that the medication induces genotoxicity through the reversal of the BAX/BCL2 ratio, increased levels of p38 MAPK, and impairment of the antioxidant system [34]
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In this study, we focused on GBM cells and found that intracellular C5aR1 promotes GBM development by increasing GPX4 expression and thereby preventing ferroptosis in a GPX4-dependent manner
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Long-term use of vitamin E-coated polysulfone membrane reduces oxidative stress markers in haemodialysis patients
