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Mitochondrial dysfunction in autism spectrum disorders: A population-based study
Es wird vermutet, dass TMAO den Cholesterintransport aus der Zelle unterdrckt

Bast 2000)

Therefore, individuals who are homozygous or compound heterozygous for an MCAD mutation can also generate abnormal MCAD proteins and inefficient enzymatic activity to metabolize medium-chain fatty acids
Zamani M, Pahlavani N, Nikbaf-Shandiz M, Rasaei N, Ghaffarian-Ensaf R, Asbaghi O, Shiraseb F, Rastgoo S

Alternatively, the final product of peroxisomal -oxidation can be used by other metabolic pathways [67, 68, 73]
