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BPC-157 accelerates axonal regeneration, while TB-500 supports neuronal survival and reduces neuroinflammation

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Nat Rev Gastroenterol Hepatol 14 , 921 (2017)

The peptide's exceptional selectivity for senescent cells, combined with its well-characterized mechanism of FOXO4-p53 interaction disruption, provides researchers with powerful capabilities for investigating senescence biology that were previously impossible to study

IGF1R signalling and its inhibition