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Downstream pathological effects driven by the E-P-OS network in uterine fibroids Persistent hormonal imbalance and the OS state within the E-P-OS network promote the development of uterine fibroids not only by directly promoting cell proliferation, inhibiting apoptosis, and enhancing the fibrotic process

PL appears most central to these effects since inactivation of PL disrupts immunization effects on both social and shuttle box behavior, whereas IL inactivation only blocks immunization effects on social behavior [180]

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Zinc concentrations were highest in sample A, while lower values were noted in samples K, B, and C