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Intracranial atherosclerosis as a contributing factor to Alzheimers disease dementia
[DOI] [Google Scholar] Perlikowska W
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3) [7], thereby reducing de novo hepatic lipogenesis and preventing hepatic fat accumulation
(A, B) Clusters of differentially accessible regions identified in Figure 1A were queried for (A) H3K27ac and (B) H3K4me1 ChIP-seq signal in human monocytes after 0h, 1h, 4h, and 24h of BDG exposure
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