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Description
Interestingly, ABHD5 mutations causative in CDS do not co-localize to LDs and are unable to stimulate PNPLA2 lipolytic activity (30), demonstrating an important function for the interaction in the etiology of CDS and lipid storage
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Its selective interaction with 3-adrenergic pathways, combined with minimal engagement of classical growth hormone signaling, allows researchers to investigate metabolic processes with a high degree of pathway specificity in laboratory and preclinical models
PMID: 40789979
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