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In addition, UCB treatment produced noticeable injury to type I spiral ganglion neurons, their axons, and terminals to cochlear inner hair cells

Immunoregulatory genes involved in T cell development, activation, signaling, and innate immune response associated with vitiligo.4,28,49,50 Variants of the PTPN22 gene have been associated with several autoimmune diseases, such as rheumatoid arthritis or systemic lupus erythematosus, making it a gene with a relevant role in the context of autoimmune diseases.61 The IKZF4 and FOXP3 genes, which play a role in gene silencing in Tregs, have also been linked.62 Several genes related to melanocyte function and survival have been invovled in vitiligo (Table 4)

159 The size and localisation of these focal injuries can vary widely, and this variability defines immediate and long-term neurological consequences

Gasperini L, Meneghetti E, Pastore B, Benetti F, Legname G (2015) Prion protein and copper cooperatively protect neurons by modulating NMDA receptor through S-nitrosylation

Cellular mechanisms of fatal early-onset autoimmunity in mice with the T cell-specific targeting of transforming growth factor-beta receptor

Alicia Zellmer, MD, and Joy Liu, MD, are resident physicians in internal medicine and members of the ABC News Medical Unit.