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Recent studies have highlighted METH-induced alterations in the hippocampus, such as reduced STX17 expression, impaired autophagosome maturation, and cognitive decline ( 2.3 Manifestations of nervous system injury METH abuse induces neurological damage that manifests as cognitive impairment, psychiatric symptoms, and motor dysfunction, with clear distinctions between the acute and chronic phases of use

This was done with brain images for both the control (saline) group and treated (BPC 157) group of rats at same intervals after the application and at the time of sacrifice

Therefore, current therapeutic strategies are mainly focused on these pathological changes

Figure 5 Downstream cellular mediators of Dihexa protection Activation of the receptor tyrosine kinase c-Met leads to the recruitment of many signaling mediators and ultimately the activation of downstream signals, most notably Akt-TOR and Ras-ERK (Organ and Tsao, 2011)

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Treatment of Tyramine Side Effects: Regardless of the initial severity, the pressor episodes evoked by tyramine are clinically transient and resolve spontaneously within 2 to 4 hours as the displaced norepinephrine is naturally cleared from the synaptic cleft and metabolized (Gillman, 2017b)