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According to the work of Stamler et al, (1993), homocysteine auto-oxidation might prevent thiol nitrosation that, under normal physiologic conditions, sustains the antiplatelet and vasorelaxing activities of NO by increasing the stability of NO (Simon et al, 1993
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Rictor/mTORC2 loss in the Myf5 lineage reprograms brown fat metabolism and protects mice against obesity and metabolic disease
