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suggested that PCA could modulate the sphingosine kinase (SphK)/S1P system and related signaling pathways to exert its anti-inflammatory effects in TNBS models of IBD [359]

Differentiation of substrate and non-substrate inhibitors of transport system xc(): an obligate exchanger of L-glutamate and L-cystine

Conversely, these organoids also exhibited upregulation of a cluster of genes which included GCLC, SAT1, FTL, HMOX1, SLC39A14 TP53 and SAT2 (Fig

Nishino, K

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The risk/benefit ratio needs to be carefully evaluated in order to provide the best treatment option to every individual patient