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This translational gap can be attributed to interspecies differences, the complexity and heterogeneity of disease models, and the limited ability of current models to fully reproduce the multifactorial nature of BIPN in patients with multiple myeloma

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5 Conclusion Considering the answers we have received from the two above interrogated AI models (and presented in the Supplementary Annexes 2, 4 contained in the related Supplementary materials), we reckon they still cannot achieve systematic literature reviews at rigor standards at specially regarding high quality identified papers comparable to those fulfilled by HI PRISMA

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