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Prx II-knockdown HaCaT cells were susceptible to H 2 O 2 -induced apoptosis mediated by Ca 2+ release from the endoplasmic reticulum through 1,4,5-triphosphate receptors (IP3Rs), the PI3K/AKT pathway and phosphorylated GSK3 (Ser9) were significant downregulated

[DOI] [PubMed] [Google Scholar] 210.Kunkel M.W., Kirkpatrick D.L., Johnson J.I., Powis G

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Beyond surface modification, exosome-encapsulated therapeutic agents also leverage inherent biocompatibility of exosomes to cross biological barriers

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