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doi: 10.1073/pnas.052592699

Thus, NAC has multiple modes of action against acetaminophen-induced hepatotoxicity, which helps explain why NAC is beneficial, even if administered with significant delay after the overdose in patients [62] and human hepatocytes [55]

These collective findings demonstrate that VPA exposure induces systemic oxidative stress, characterized by concurrent antioxidant depletion (CAT, GSH-Px, GSH, SOD) and elevation of oxidative damage markers (MDA, T-NOS, NO)

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