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OberoiRVlacilAKSchuettJSchosserFSchuettHTietgeUJFet alAnti-tumor necrosis factor-alpha therapy increases plaque burden in a mouse model of experimental atherosclerosis

doi: 10.3389/fneur.2020.00545

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Several small-molecule inhibitors of CYP51A1 have been developed, including clinically used azoles such as fluconazole and ketoconazole, which were originally designed to target fungal CYP51A1 but also inhibit the human enzyme (Table 3)