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Furthermore, the effect was more pronounced in rats who were administered the lowest dose (0.5 g/kg) and it decreased with the increase in dose [10]

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Finally, we used to model to show how the observed metabolic profile of Down syndrome could arise from trisomy 21 and moderate oxidative stress and how the observed metabolic profile of autism patients could arise from high oxidative stress and increased adenosine concentration
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When BPC-157 activates this pathway, fibroblasts (the cells responsible for collagen production) migrate more efficiently to injury sites and demonstrate improved survival under oxidative stress conditions common in damaged tissue

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